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CIED programming considerations during and after an AVN ablation

Pre ablation check

Perform normal check

Ensure all leads (most important V lead) are functioning well

•No sudden change in impedance, sensing, threshold

Ensure wound is healed well

•No hematoma, evidence of infection, etc 

Reprogram to an appropriate mode for ablation

•Single chamber:  VVI 30/40

•Dual chamber:  VVI 30/40

AVN ablation procedure

1 sheath in femoral vein for ablation catheter +-2nd sheath if backup RV pacing catheter is used

Ablation catheter can be either non irrigated or irrigated, 4mm tip or 8mm tip (physician preference)

Ideally the AVN is ablated as opposed to the HIS - leaving a junctional escape rhythm

  • Diagrammatic view of the atrial septum in the right atrium to identify the various atrial nodal inputs to the atrioventricular (AV) node and His bundle. The fast pathway (yellow) runs along the anterior limbus of the oval fossa to enter the AV node (blue triangle), at the apex of which is the His bundle (red dot). The slow pathway runs from the coronary sinus (green area) to the AV node, whereas the central transitional cells traverse the area between the slow and fast pathways to input to the AV node.

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See rapid firing for the AV node during ablation follow by complete heart block

fast slow.png

Potential risk of pacing inhibition during ablation

•Notify physician if this occurs

•Consider ASYNC if persistent

inhib.jpg

Post ablation check

Perform normal check

Ensure no changes in testing values

•Possible risk of lead damage during ablation

•Possible risk of moving lead with catheters

Ensure ablation has worked with VVI 40 / sensing testing

•In some circumstances when patient is in sinus rhythm, brief periods of AAI pacing may be required to prove CHB

Post ablation concerns

Increase in sympathetic activity post AVN ablation


Well documented risk of ventricular arrhythmias in the 1st few months following AVN ablation

•Particularly Torsades de Pointes


Post AVN ablation results in increased sympathetic nerve activity, increased cardiac action potential duration and increased effective refractory period.

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Post ablation programming

Research has shown pacing at faster rates (e.g., 80 or 90 ppm) immediately post AVN ablation compared to standard rates (60 ppm), decreases sympathetic nerve activity

Ensure no algorithm is turned on which may violate the lower rate limit, as this may result in a long-short sequence

•The perfect recipe for Tosades



ICD therapies

•?SVT discriminators?

Poor programming example

Patient had a single chamber SJM PPM inserted prior to planned AVN ablation.

Was programmed to VVI 40 prior to ablation

V autocapture was turned ON to preserve battery and allow for trends of threshold

Had successful, uncomplicated AVN ablation

Device was reprogrammed to VVIR 80-120

Called ambulance due to persistent episodes of syncope and pre-syncope

Presenting rhythm

tdppppp.png

When V auto threshold is turned on in a SJM, if the device is a single chamber, or is a dual chamber with single chamber mode programming (VVI), a hysteresis automatically turns ON.

•Set 10ppm below current LRL.

•For this case mode was VVI 40, hysteresis goes to 30

The hysteresis rate was not turned OFF or changed during or at the end of the ablation

•VVIR 80-120

•Hysteresis 30

Algorithms that may violate LRL

Hysteresis

MVP modes

AV searches

Some autocapture tests (with no backup pulse)

NCAP

Rest rate/sleep rate

Post AVN ablation follow up

Book ~6 week post AVN ablation appointment with follow up clinic

Demonstrate whether the procedure was a success

•If recurrent, book for repeat ablation

Rates typically slowly titrated down (e.g., from 80 to 70)

Turn off all SVT discriminators

After 3 months consider restoring autocapture settings if they were turned off.

Post AVN ablation complications

APO post AVN ablation – immediately or shortly after (up to 6 weeks post)


Studies have shown that this generally occurs due to worsening of MR


Usually due to the dilatation of the mitral valve annulus

•Due to slower rate

•Can be due to increased LV end
diastolic diameters (does not occur
in every pt)

lungs.jpg
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